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primary antibodies specific for bmp2 mouse monoclonal ag13501  (Proteintech)


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    Proteintech primary antibodies specific for bmp2 mouse monoclonal ag13501
    Primary Antibodies Specific For Bmp2 Mouse Monoclonal Ag13501, supplied by Proteintech, used in various techniques. Bioz Stars score: 90/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
    https://www.bioz.com/product/bmp2+mouse+monoclonal+antibody/anti+bmp2/pm39871679-293-29-33
    Average 90 stars, based on 1 article reviews
    primary antibodies specific for bmp2 mouse monoclonal ag13501 - by Bioz Stars, 2026-10
    90/100 stars

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    Article Title: Caffeic Acid Phenethyl Ester Enhances Bone Repair-related Factors in MC3T3-E1 Cells.
    Article Snippet: Apical periodontitis is an inflammatory disease caused by bacterial infection in the root canal that spreads to the apical periodontal tissues, resulting in bone resorption around the root apex as the disease progresses.. Vascular endothelial growth factor (VEGF), a growth factor involved in angiogenesis, plays an important role in bone remodeling.. We reported that caffeic acid phenethyl ester (CAPE), a bioactive substance of propolis, induces VEGF in odontoblast-like cells and dental pulp cells.



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    Image Search Results


    Expression of osteogenesis markers in xeno-free 3D spheroids. (A) Relative expression (fold changes) of osteogenesis-related genes after 7 days in 2D and 3D GPCs and BMSCs under non-induced (–) and osteogenically induced conditions (+). Data represent means ( n = ≥ 3 experimental replicates); statistical analyses are based on delta-Ct values; *p < 0.05; **p < 0.001. (B) IF staining of BMP2 and OCN in 3D spheroids after 14 days of suspension culture; cell nuclei are stained in DAPI: scale bars 100 μm.

    Journal: Frontiers in Bioengineering and Biotechnology

    Article Title: Xeno-Free Spheroids of Human Gingiva-Derived Progenitor Cells for Bone Tissue Engineering

    doi: 10.3389/fbioe.2020.00968

    Figure Lengend Snippet: Expression of osteogenesis markers in xeno-free 3D spheroids. (A) Relative expression (fold changes) of osteogenesis-related genes after 7 days in 2D and 3D GPCs and BMSCs under non-induced (–) and osteogenically induced conditions (+). Data represent means ( n = ≥ 3 experimental replicates); statistical analyses are based on delta-Ct values; *p < 0.05; **p < 0.001. (B) IF staining of BMP2 and OCN in 3D spheroids after 14 days of suspension culture; cell nuclei are stained in DAPI: scale bars 100 μm.

    Article Snippet: The primary antibodies rabbit polyclonal anti-SOX2 (Abcam, dilution 1:1000), mouse monoclonal anti-BMP2 (Bio-Techne, Abingdon, United Kingdom, dilution 1:100), and rabbit polyclonal anti-OCN (Abcam, dilution 1:100) were incubated ON at 4°C.

    Techniques: Expressing, Staining, Suspension

    Histopathological profiles in the groups

    Journal: Vascular medicine (London, England)

    Article Title: Incremental Effects of Diabetes Mellitus and Chronic Kidney Disease in Medial Arterial Calcification: Synergistic Pathways for Peripheral Artery Disease Progression

    doi: 10.1177/1358863X19842276

    Figure Lengend Snippet: Histopathological profiles in the groups

    Article Snippet: Primary antibodies against the following antigens were used: rabbit polyclonal a-SMA (PA1–37024-Thermofisher, IL, 1:100 dilution), mouse monoclonal a-SMA (FITC-F3777-Sigma Aldrich, MO, 1:500 dilution), mouse monoclonal BMP2 (ab6285-Abcam, MA, 1:100 dilution), and Fetuin-A (NBP1–91192 -Novus Biologicals, CO, 1:200 dilution).

    Techniques: Staining, Expressing

    BMP-2 density (A-D): Immunostaining for BMP-2 using diaminobenzene brown chromogen labeling, showing BMP-2 expression in the intimal and medial layers of peripheral atherosclerotic plaques from patients without DM or CKD (A), DM only (B), CKD only (C) and DM+CKD (D) (20 X magnification, scale bar=50μm). E) Scatter gram shows results of quantitative analysis of BMP-2 protein density levels in peripheral plaques among groups, No-DM/no-CKD, (n=5); DM(n=6); CKD (n=6); DM+CKD (n=8): (n = observations/high power field); Univariate Analysis of Variance with Bonferroni’s post-hoc test. *** p<0.001 vs. no-DM/no-CKD. F-I)

    Journal: Vascular medicine (London, England)

    Article Title: Incremental Effects of Diabetes Mellitus and Chronic Kidney Disease in Medial Arterial Calcification: Synergistic Pathways for Peripheral Artery Disease Progression

    doi: 10.1177/1358863X19842276

    Figure Lengend Snippet: BMP-2 density (A-D): Immunostaining for BMP-2 using diaminobenzene brown chromogen labeling, showing BMP-2 expression in the intimal and medial layers of peripheral atherosclerotic plaques from patients without DM or CKD (A), DM only (B), CKD only (C) and DM+CKD (D) (20 X magnification, scale bar=50μm). E) Scatter gram shows results of quantitative analysis of BMP-2 protein density levels in peripheral plaques among groups, No-DM/no-CKD, (n=5); DM(n=6); CKD (n=6); DM+CKD (n=8): (n = observations/high power field); Univariate Analysis of Variance with Bonferroni’s post-hoc test. *** p<0.001 vs. no-DM/no-CKD. F-I)

    Article Snippet: Primary antibodies against the following antigens were used: rabbit polyclonal a-SMA (PA1–37024-Thermofisher, IL, 1:100 dilution), mouse monoclonal a-SMA (FITC-F3777-Sigma Aldrich, MO, 1:500 dilution), mouse monoclonal BMP2 (ab6285-Abcam, MA, 1:100 dilution), and Fetuin-A (NBP1–91192 -Novus Biologicals, CO, 1:200 dilution).

    Techniques: Immunostaining, Labeling, Expressing

    SMCs Expressing BMP2 Immunofluorescence staining for α-SMA (red) and BMP2 (green) in peripheral atherosclerotic plaques with MAC from patients without DM or CKD (A), DM only (B), CKD only (C) and DM+CKD (D); SMCs positive for BMP2 visualized in yellow (20 X magnification, scale bar=20μm). E) Scatter gram shows results of quantitative analysis of SMCs (α-SMA+) expressing BMP2 in peripheral atherosclerotic plaques among groups; No-DM/no-CKD, (n=8); DM(n=13); CKD (n=9); DM+CKD (n=16): (n = observations /high power field); Univariate Analysis of Variance with Bonferroni’s post-hoc test. * p<0.05, *** p<0.001 vs. no-DM/no-CKD.

    Journal: Vascular medicine (London, England)

    Article Title: Incremental Effects of Diabetes Mellitus and Chronic Kidney Disease in Medial Arterial Calcification: Synergistic Pathways for Peripheral Artery Disease Progression

    doi: 10.1177/1358863X19842276

    Figure Lengend Snippet: SMCs Expressing BMP2 Immunofluorescence staining for α-SMA (red) and BMP2 (green) in peripheral atherosclerotic plaques with MAC from patients without DM or CKD (A), DM only (B), CKD only (C) and DM+CKD (D); SMCs positive for BMP2 visualized in yellow (20 X magnification, scale bar=20μm). E) Scatter gram shows results of quantitative analysis of SMCs (α-SMA+) expressing BMP2 in peripheral atherosclerotic plaques among groups; No-DM/no-CKD, (n=8); DM(n=13); CKD (n=9); DM+CKD (n=16): (n = observations /high power field); Univariate Analysis of Variance with Bonferroni’s post-hoc test. * p<0.05, *** p<0.001 vs. no-DM/no-CKD.

    Article Snippet: Primary antibodies against the following antigens were used: rabbit polyclonal a-SMA (PA1–37024-Thermofisher, IL, 1:100 dilution), mouse monoclonal a-SMA (FITC-F3777-Sigma Aldrich, MO, 1:500 dilution), mouse monoclonal BMP2 (ab6285-Abcam, MA, 1:100 dilution), and Fetuin-A (NBP1–91192 -Novus Biologicals, CO, 1:200 dilution).

    Techniques: Expressing, Immunofluorescence, Staining